2004;350:1189C99

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2004;350:1189C99. and bone tissue deformities in OPG-knockout mice. These results stem through the unopposed RANKL activity due to the lack of a decoy receptor. OPG-knockout mice will be the initial pet model for learning osteoporosis. As a result, OPG can be an essential harmful regulator of osteoclastogenesis. Another molecule that stimulates osteoclastogenesis is certainly M-CSF. […]